Tibia |
Robinson
(2021)[@140576] |
Patients w/ >20% glenoid bone loss and evidence of an off-track lesion and w/out neurologic injury or seizure disorder |
Provencher
(2019)[@140573] |
Patients w/ failed Latarjet and w/out prior neurologic injury, seizure disorder, bone graft requirements to humeral head, or findings of multidirectional or posterior instability |
Frank
(2018)[@140574] |
Patients with >15% glenoid bone loss (patients were recommended DTA vs. Latarjet if bone loss > 25% or significant cartilage component or if had failed prior Latarjet) and w/out systemic hyperlaxity, ipsilateral arm neurologic injury, or posterior and/or multidirectional instability |
Provencher
(2017)[@140575] |
Patients with ≥ 15% anterior glenoid bone loss w/out systemic hyperlaxity, ipsilateral arm neurologic injury, posterior and/or multidirectional instability |
Coracoid |
Belangero
(2021)[@140617] |
Patients with high-demand sports participation (>7hr/week), 10-20% glenoid bone loss, having primary surgery w/out RCT or SLAP |
Chillemi
(2021)[@140603] |
Patients w/out multidirectional or volitional instability or hypermobility syndrome or rotator cuff pathology at latest FU and minimum 24 years FU |
Marjanovic
(2021)[@140616] |
Patients w/ glenoidal bone loss greater than 13.5% and engaging Hill–Sachs lesion or previous failed arthroscopic Bankart repair and w/out hyperlaxity, infection in progress or neurological involvement of axillary nerve |
Rai
(2021)[@140608] |
Patients having primary surgery, glenoid loss >20% or significant Hill Sach’s lesion |
Sinha
(2021)[@140646] |
Patients w/ off-tract bipolar bone loss, glenoid loss >20%, glenoid loss <20% but an ISIS >3, a history of sleep dislocations, and w/out habitual dislocation, generalized laxity of joints, an associated fracture, bilateral involvement of shoulder, a history of failed surgery, osteoarthritic changes, or neuromuscular involvement |
Thon
(2021)[@140578] |
Patients w/ a seizure disorder and subcritical bone loss having primary surgery |
Patients w/out a seizure disorder and subcritical bone loss having primary surgery |
Ali
(2020)[@140610] |
Patients aged >18 years w/ anteroinferior glenoid osteochondral defect >13.5%, ISIS >3 combined and mid-range positive anterior apprehension |
Cohen
(2020)[@140585] |
Patients w/ anterior shoulder instability w/ preop and postop CT scan and w/out major deviation from the most commonly used technique (such as the congruent arc technique), those that had to be reoperated for hardware removal, or those in which the graft did not heal promptly with no or minor bone reabsorption |
Patients w/ anterior shoulder instability w/ preop and postop CT scan and w/out major deviation from the most commonly used technique (such as the congruent arc technique), those that had to be reoperated for hardware removal, or those in which the graft did not heal promptly with major or total bone reabsorption |
Di Giacomo
(2020)[@140650] |
Patients having primary Latarjet, ≥2 confirmed dislocations, ISIS ≥4 and w/out previous surgical procedure, RCT, shoulder pain w/out instability or voluntary and/or multidirectional instability |
Domos
(2020)[@140581] |
Patients aged <17 years w/ unfused glenoid and humeral ossification centers, w/out previous surgery, RCT, accompanying fractures of the humerus, neurologic deficit, or incomplete outcome data |
Elamo
(2020)[@140595] |
Patients w/ previous failed arthroscopic Bankart |
Maman
(2020)[@140652] |
Patients having primary surgery and age ≤40 w/ minimum FU 5 years w/out previous shoulder surgery or other injuries in affected shoulder (e.g., a superior labral anterior-posterior lesion, RCT, cartilage defect, humeral avulsion of the glenohumeral ligament, voluntary dislocation or multidirectional instability) |
Rossi
(2020)[@140618] |
Athletes having classic Latarjet w/ glenoid bone defect >20% on CT having primary surgery w/out other types of instability (e.g., posterior, or voluntary) |
Athletes having congruent arc Latarjet w/ glenoid bone defect >20% on CT having primary surgery w/out other types of instability (e.g., posterior or voluntary) |
Werthel
(2020)[@140591] |
Patients who read/speak French having revision surgery w/out additional shoulder pathology (posterior or multidirectional instability, pathological involvement of the long head of the biceps, RCT, or symptomatic acromioclavicular joint arthritis) |
Patients who read/speak French having primary surgery w/out additional shoulder pathology (posterior or multidirectional instability, pathological involvement of the long head of the biceps, RCT, or symptomatic acromioclavicular joint arthritis) |
Yapp
(2020)[@140593] |
Patients w/ >20% glenoid bone loss or evidence of an engaging Hill-Sachs lesion having primary surgery |
Patients w/ >20% glenoid bone loss or evidence of an engaging Hill-Sachs lesion having revision surgery |
De Carli
(2019)[@140657] |
Athletes w/ 6+ years FU, inclusion in sports activities even if not professional, 2-10 dislocations, w/out glenoid bone loss, multidirectional instability, voluntary dislocation, previous surgery on affected shoulder; preoperative signs of glenohumeral osteoarthritis, association of RCT or SLAP lesions, or concomitant systemic disease (i.e. lupus, arthritis) |
Ernstbrunner
(2019)[@140582] |
Patients ≥40 years w/ failed soft tissue repair w/out multidirectional instability, chronic dislocation, dislocated fracture of greater tuberosity, irreparable RCT, or convulsive disorders |
Flinkkilä
(2019)[@140592] |
Patients having primary surgery and w/out recurrent seizures |
Patients having revision of failed Bankart and w/out recurrent seizures |
Minkus
(2019)[@140597] |
Patients with previous soft tissue surgery |
Moroder
(2019)[@140568] |
Patients w/ anterior shoulder instability w/ recurrent dislocations (≥2), glenoid bone loss ≥15% and w/out concomitant shoulder pathologies, previous surgical procedures except for Bankart, neuromuscular pathologies including seizure disorders, history of infection |
Xu
(2019)[@140661] |
Patients w/ dislocation of shoulder joint w/ slight external force, for ≥1 year, ISIS score ≥3, higher sports requirement (especially over-shoulder movement) and w/out condition other than osteoarthritis of the shoulder (significant changes in joint space), multiple recurrent shoulder subluxations or dislocations, severe epilepsy, unclosed osteoepiphysis, severe glenoid bone loss (glenoid loss of contour on anteroposterior radiograph), active infection, and a major medical illness. |
Bah
(2018)[@140586] |
Patients having primary surgery w/ significant Hill Sachs defect (>30%), ISIS >3, w/out significant glenoid bone defect (>30%), RCT, instability in multiple directions, voluntary instability, or previous surgery on shoulder |
Baverel
(2018)[@140663] |
Professional athletes aged 16-30 w/ no previous surgery, w/ positive apprehension test in cocking position, ISIS >2, evidence of anterior instability lesions (humeral Hill-Sachs and/or glenoid-sided defects) |
Recreational athletes aged 16-30 w/ no previous surgery, w/ positive apprehension test in cocking position, ISIS >2, evidence of anterior instability lesions (humeral Hill-Sachs and/or glenoid-sided defects) |
de l’Escalopier
(2018)[@140580] |
Soldiers w/ >15 years FU and w/out associated injuries or complementary procedures or any history related to operated-on shoulders |
Erşen
(2018 KSSTA)[@140666] |
Patients w/ subscapularis tenotomy and w/out bilateral surgery |
Patients w/ subscapularis split and w/out bilateral surgery |
Frank
(2018)[@140574] |
Patients with >15% glenoid bone loss and w/out systemic hyperlaxity, neurologic injury, posterior and/or multidirectional instability |
Jeon
(2018)[@140613] |
Patients with 15-20% glenoid bone defect having primary surgery and w/out a bony Bankart lesion or engaging Hill-Sachs lesion |
Kawasaki
(2018)[@140589] |
Rugby players w/ injury involving acute or recurrent dislocation or subluxation and w/out any other shoulder surgery |
Kee
(2018)[@140669] |
Non-collision athletes (basketball, baseball, swimming, badminton, volleyball, taekwondo, tennis) |
Collision athletes (soccer, judo, boxing, rugby, wrestling, martial arts) |
Lateur
(2018)[@140604] |
(No inclusion or exclusion criteria reported) |
Moroder
(2018 JSES)[@140555] |
Patients >40 years w/ anterior glenoid bone defects and/ or the presence of an irreparable yet functionally compensated RCT |
Privitera
(2018)[@140619] |
Contact and collision sports athletes w/ signs of anterior instability, clinically relevant glenoid osseous defect as measured on CT, or prior failed anterior shoulder stabilization surgery and w/out posterior or multidirectional instability, an irreparable RCT or one requiring repair, pathological involvement of the biceps tendon or acromioclavicular joint requiring surgical intervention, or a posterior labral tear requiring repair |
Ranalletta
(2018 JARS)[@140598] |
Athletes w/ failed operative stabilization and lesion of the anterior aspect of the glenoid rim >20% and w/out posterior or voluntary instability |
Ranalletta
(2018 AJSM)[@140598] |
Competitive rugby players (practice >2 times per week and competition during weekends) and glenoid bone defect >20% shown on CT w/out other types of instability (e.g., posterior, or voluntary) |
Rossi
(2018)[@140674] |
Competitive athletes (practice >2 times/week and competition during weekends) w/ glenoid bone defect >20% on CT having primary surgery and w/out other types of instability (e.g., posterior or voluntary) |
Competitive athletes (practice >2 times/week and competition during weekends) w/ glenoid bone defect >20% having revision surgery |
Yang
(2018)[@140605] |
Patients w/ <25% glenoid bone loss, engagement of the Hill-Sachs lesion and/or off-track Hill-Sachs lesion w/out symptomatic multidirectional instability or RCT |
Sub-group: revision patients |
Sub-group: >15% glenoid bone loss |
Sub-group: collision and contact athletes |
Cautiero
(2017)[@140558] |
Patients involved w/ competitive contact sport or sport w/ forced overhead activity, glenoideal bone loss >15% or Hill-Sachs >⅓, capsular lesion or HAGL lesion, excessive thinness of capsular tissue, failure of arthroscopic stabilization or dislocation w/ glenoid fracture and w/out glenoid fracture >⅓ or subscapularis rupture |
Elganainy
(2017)[@140641] |
Patients w/Hill-Sachs lesions >20% but <40%, ≥3 dislocations, engaging Hill-Sachs lesion found during arthroscopy, w/out atraumatic, multidirectional or voluntary instability |
Erşen
(2017 OTSR)[@140577] |
Patients w/ epilepsy |
Patients w/out epilepsy |
|
Gough
(2017)[@140602] |
Patients w/ evidence of glenoid bone deficiency >25%, an engaging Hill-Sachs lesion, or a previously failed arthroscopic or open anterior capsular procedure. |
Kee
(2017)[@140642] |
Patients w/ significant glenoid bone loss on pre op CT or previous failed instability operation |
Kordasiewicz
(2017)[@140643] |
Patients having primary stabilization surgery |
Marion
(2017)[@140567] |
N/A |
Vadala
(2017)[@140644] |
Patients ≤50 involved in sports activities as amateurs w/out RCT, multidirectional instability, concomitant systemic disease, such as lupus or rheumatoid arthritis |
Zhang
(2017)[@140645] |
Patients undergoing classic Bristow-Latarjet w/ glenoid defect >10% and w/out pre op shoulder osteoarthritis, multidirectional instability, severe uncontrollable epilepsy, chronic locked anterior shoulder dislocation, or bilateral dislocation |
Patients undergoing Bristow-Latarjet w/ coracoid osteotomy and bone fixation position parameter measured through simulating a 3D congruent arc glenoid reconstruction model) w/ >10% glenoid defect and w/out pre op shoulder osteoarthritis, multidirectional instability, severe uncontrollable epilepsy, chronic locked anterior shoulder dislocation, or bilateral dislocation |
Beranger
(2016)[@140647] |
Athletes <50 year and ISIS >3 |
Blonna
(2016)[@140648] |
Patients w/out previous surgery, RCT or SLP, or epilepsy |
Chaudhary
(2016)[@140594] |
Patients w/ previous failed Bankart repair or positive apprehension test and w/out a primary Latarjet |
Cho
(2016)[@140611] |
Patients w/ ISES ≥ 3 points or large Hill-Sachs lesion confirmed to be engaging over glenoid rim and w/out significant glenoid bone loss (glenoid deficit <25% of the inferior glenoid diameter), rotator cuff tears, hyperlaxity (2+ or greater laxity on the sulcus sign), voluntary instability or severe pre-existing osteoarthritis |
Jamal
(2016)[@140649] |
Patients w/recurrent dislocation, subluxation or painful shoulder |
Khater
(2016)[@140609] |
Patients>16 years w/ clear history of tramadol abuse, tramadol dose per day >200 mg, abuse duration >2 months and w/out associated shoulder abnormality |
Li
(2016)[@140587] |
Patients w/ chronic locked anterior shoulder dislocation (>3 months, irreducible at admission), glenoid defect of <40% and w/out previous surgical procedure congenital shoulder dislocation, concomitant 3 or 4-part proximal humeral fracture, acromioclavicular dislocation, distal clavicle fracture, or other scapular fractures (except Bankart lesions), or neurovascular injury |
Ropars
(2016)[@140651] |
Patients w/ unilateral symptomatic involuntary unidirectional anterior shoulder instability having primary stability surgery w/ positive anterior apprehension sign in maximal abduction and ER and an ISIS ≥4 w/out RCT or posterior instability |
Venkatachalam
(2016)[@140653] |
Antero-inferior glenoid bone loss >20% w/ or w/o large Hill–Sachs lesion or w/ previous Bankart, or w/ high-risk (involved in high-energy contact sports) w/ any glenoid bone loss |
Abelhady
(2015)[@140654] |
Patients w/ ≥20 recurrences and generalized ligamentous laxity and Hill-Sachs lesion <20% of head diameter w/ no glenoid defect |
Arianjam
(2015)[@140655] |
High risk athletes w/ antero-inferior glenoid bone loss, ongoing history of pain, and apprehension or instability |
Balestro
(2015)[@140560] |
(No additional inclusion/exclusion criteria noted) |
Dos Santos
(2015)[@140656] |
Patients having revision w/ positive apprehension test or recurrent dislocations after Bankart's repair |
Flinkkilä
(2015)[@140596] |
Patients w/ previous failed Bankart |
Gordins
(2015)[@140588] |
Patients w/ 33-35 years FU |
Ikemoto
(2011)[@140590] |
Patients w/ bone loss >25% in the anteroinferior margin of the glenoid cavity |
Moon
(2015)[@140658] |
Patients w/ significant glenoid bone loss (glenoid deficit ≥25%) on CT or previous failed instability operation and w/out an ISIS ≥3 or a large Hill-Sachs lesion that was confirmed to be engaging over the glenoid rim during arthroscopy, only soft tissue lesion, tear of the rotator cuff tendon, hyperlaxity or voluntary instability; or severe pre-existing osteoarthritis |
Ruci
(2015)[@140607] |
NR |
Yang
(2016)[@140659] |
Patients w/ engaging Hill-Sachs by examination confirmed with arthroscopy, and >25% anterior glenoid bone loss w/out symptomatic multidirectional instability |
Zimmermann
(2016)[@140660] |
Patients having primary surgery w/out posterior or multidirectional instability, convulsive disorders, or massive RCT |
Tasaki
(2015)[@140606] |
Competitive rugby players who were participating at or above the high school level and who continued to actively participate in the sport for more than 2 years |
Zhu
(2015)[@140554] |
Patients with traumatic recurrent anterior shoulder instability and >20% glenoid defect having primary surgery and w/out multidirectional instability, nonunion or implant failure, or incomplete radiologic evaluation |
Bessière
(2014)[@140662] |
Patients w/out other shoulder stabilization procedures, voluntary instability, acute instability, unstable painful shoulder, associated epilepsy, or associated RCT |
Bouju
(2014)[@140664] |
Patients w/out RCT |
Mizuno
(2014)[@140665] |
Patients w/ long term FU (minimum 18 years) w or w/out hyperlaxity and w/out previous repair, ‘‘subtle’’ anterior instability, Bankart lesion (painful shoulder in the throwing athlete), voluntary habitual anterior instability |
Atalar
(2013)[@140601] |
Patients w/ glenoid bone loss |
Bessière
(2013)[@140667] |
Patients w/out prior surgery, voluntary or posterior instability, rotator cuff lesions and painful shoulders due to unidentified episode of instability |
Di Giacomo
(2013)[@140668] |
Patients w/ ISIS ≥6, w/ or w/out ligamentous hyperlaxity, and w/out RCT, previous surgery, or multidirectional instability |
Holzer
(2013)[@140612] |
Patients w/ ≥25% glenoid bone loss, an engaging Hill-Sachs lesion, or failed anterior labral repair with glenoid bone loss w/out voluntary subluxation or dislocation |
Lädermann
(2013)[@140614] |
Patients having primary surgery with long term follow-up (≥10 years) |
Aydin
(2012)[@140632] |
Patients w/out bony Bankart lesions |
Cerciello
(2012)[@140584] |
Soccer players
(7 goalkeepers, 19 general population) |
Neyton
(2012)[@140670] |
Rugby players undergoing primary stabilization procedure w/out other types of instability (eg, posterior or voluntary) |
Paladini
(2012)[@140671] |
NR (Only specifies patients w/ anterior shoulder instability) |
Raiss
(2012)[@140579] |
Patients with epilepsy |
Schmid
(2012)[@140599] |
Patients w/ previous stabilizations (other than Latarjet) and instability recurrence associated w/ lesion of anterior aspect of glenoid rim on CT w/ a craniocaudal extension of at least one-third of maximal anteroposterior diameter of glenoid and w/out fatty infiltration of subscapularis muscle >stage II |
Shah
(2012)[@140672] |
Patients w/ glenoid bone loss of <30% or prior arthroscopic or open capsule and labral (Bankart) repair and w/out symptoms of multidirectional glenohumeral instability, volitional instability, or prior arthroplasty |
Emami
(2011)[@140673] |
Non-athletes w/out posterior instability, multidirectional instability, RCT, or impingement syndrome. |
Hovelius
(2011)[@140675] |
Patients w/ traumatic anterior shoulder instability w/out multidirectional instability |